Dihydromyricetin has a significant protective effect on carbon tetrachloride induced liver injury in cultured rat liver cells, as well as D2 galactosamine and lipopolysaccharide induced liver injury in mice. Dihydromyricetin can protect the liver, accelerate the rapid decomposition of ethanol metabolite acetaldehyde, turn it into a non-toxic substance, and reduce its damage to liver cells. In addition, dihydromyricetin can improve the increase in serum lactate dehydrogenase activity caused by liver cell damage, inhibit the formation of collagen fibers in hepatic M cells, and thus play a protective role in the liver, significantly reducing the damage of ethanol to the liver, and quickly restoring the normal state of the liver. Microherb experiments have shown that dihydromyricetin can protect the liver, accelerate the rapid decomposition of ethanol metabolite acetaldehyde, and become non-toxic substances, reducing damage to liver cells. Dihydromyricetin has a rapid and long-lasting effect, making it a good product for protecting the liver, relieving alcohol and sobering up.